Peer-reviewed publications

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Vinge, L., Lees, G. E., Nielsen, R., Kashtan, C. E., Bahr, A. & Christensen, E. I. (2010). The effect of progressive glomerular disease on megalin-mediated endocytosis in kidney. Nephrology Dialysis Transplantation, 25, 2458-2467.

Vincenzo de Paoli, F., Media, A. S., Juhl-Olsen, P., Katballe, N., Vad, H., Petersen, R. H., Højsgaard, A. & Christensen, T. D. (2024). High haller index and outcomes in pectus excavatum repair: What to believe in this matter? The Journal of Thoracic and Cardiovascular Surgery, 168(5), e176-e177. https://doi.org/10.1016/j.jtcvs.2024.05.001

Vimaleswaran, K. S., Cavadino, A., Berry, D. J., Jorde, R., Dieffenbach, A. K., Lu, C., Alves, A. C., Heerspink, H. J. L., Tikkanen, E., Eriksson, J., Wong, A., Mangino, M., Jablonski, K. A., Nolte, I. M., Houston, D. K., Ahluwalia, T. S., van der Most, P. J., Pasko, D., Zgaga, L. ... LifeLines Cohort Study investigators (2014). Association of vitamin D status with arterial blood pressure and hypertension risk: a mendelian randomisation study. The Lancet Diabetes & Endocrinology, 2(9), 719-29. https://doi.org/10.1016/S2213-8587(14)70113-5

VILSEN, B. & ANDERSEN, J. P. (1987). Effect of phospholipid, detergent and protein‐protein interaction on stability and phosphoenzyme isomerization of soluble sarcoplasmic reticulum Ca‐ATPase. European Journal of Biochemistry, 170(1-2), 421-429. https://doi.org/10.1111/j.1432-1033.1987.tb13716.x

Vilsen, B. (1999). Mutant Phe788→Leu of the Na⁺,K⁺-ATPase is inhibited by micromolar concentrations of potassium and exhibits high Na⁺-ATPase activity at low sodium concentrations. Biochemistry, 38, 11389-11400.

Vilsen, B., Holm, R. & Nielsen, H. N. (2022). A novel super-efficient rescue mutation restoring the functional capability of Na,K-ATPase α3 in neurological disease. 58-59. Poster session presented at 16th International Conference on NA,K-ATPase and Related Transport ATPases, Banff, Canada.

Vilsen, B., Andersen, J. P., Petersen, J. & Jørgensen, P. L. (1987). Occlusion of 22Na+ and 86Rb+ in membrane-bound and soluble protomeric alpha beta-units of Na,K-ATPase. Journal of Biological Chemistry, 262(22), 10511-10517.

Vilsen, B. & Andersen, J. P. (1986). Occlusion of Ca²⁺ in soluble monomeric sarcoplasmic reticulum Ca²⁺-ATPase. Biochimica et biophysica acta - Biomembranes, 855(3), 429-431. https://doi.org/10.1016/0005-2736(86)90089-1

Vilsen, B., Ramlov, D. & Andersen, J. P. (1997). Functional consequences of mutations in the transmembrane core region for cation translocation and energy transduction in the Na⁺,K⁺-ATPase and the SR Ca²⁺-ATPase. Annals of the New York Academy of Sciences, 834, 297-309. https://doi.org/10.1111/j.1749-6632.1997.tb52260.x

Vilsen, B. & Andersen, J. P. (1992). Deduced amino acid sequence and E1-E2 equilibrium of the sarcoplasmic reticulum Ca²⁺-ATPase of frog skeletal muscle Comparison with the Ca²⁺-ATPase of rabbit fast twitch muscle. FEBS Letters, 306(2-3), 213-218. https://doi.org/10.1016/0014-5793(92)81003-5

Vilsen, B., Andersen, J. P. & MacLennan, D. H. (1991). Functional consequences of alterations to hydrophobic amino acids located at the M₄S₄ boundary of the Ca²⁺-ATPase of sarcoplasmic reticulum. Journal of Biological Chemistry, 266(28), 18839-18845.

Vilsen, B. & Andersen, J. P. (1992). CrATP-induced Ca²⁺ occlusion in mutants of the Ca²⁺-ATPase of sarcoplasmic reticulum. Journal of Biological Chemistry, 267(36), 25739-25743.

Vilsen, B. & Andersen, J. P. (1992). Mutational analysis of the role of Glu³⁰⁹ in the sarcoplasmic reticulum Ca²⁺-ATPase of frog skeletal muscle. FEBS Letters, 306(2-3), 247-250. https://doi.org/10.1016/0014-5793(92)81010-J

Vilsen, B. & Andersen, J. P. (1988). Use of a small TSK GSW high-performance liquid chromatographic column for large-zone chromatographic studies of monomer-oligomer equilibria of membrane protein. Journal of Chromatography A, 442(C), 229-236. https://doi.org/10.1016/S0021-9673(00)94471-6

Vilsen, B. & Andersen, J. P. (1987). Characterization of CrATP-induced calcium occlusion in membrane-bound and soluble monomeric sarcoplasmic reticulum Ca²⁺-ATPase. Biochimica et biophysica acta - Biomembranes, 898(3), 313-322. https://doi.org/10.1016/0005-2736(87)90072-1

Vilsen, B. & Andersen, J. P. (1998). Mutation to the glutamate in the fourth membrane segment of Na⁺,K⁺- ATPase and Ca²⁺-ATPase affects cation binding from both sides of the membrane and destabilizes the occluded enzyme forms. Biochemistry, 37(31), 10961-10971. https://doi.org/10.1021/bi9802925

Vilsen, B., Andersen, J. P. & MacLennan, D. H. (1992). Mutational analysis of the role of Lys684 in the Ca²⁺-ATPase of sarcomplasmic reticulum. Acta Physiologica Scandinavica, Supplement, 146(607), 279-284.

Vilsen, B., Andersen, J. P., Petersen, J. & Jørgensen, P. L. (1988). Occlusion of 22Na+ and 86Rb+ in membrane-bound and soluble protomeric alpha beta-units of Na,K-ATPase. Progress in Clinical and Biological Research, 268 A, 383-390.

Vilsen, B. & Andersen, J. P. (1992). Interdependence of Ca²⁺ occlusion sites in the unphosphorylated sarcoplasmic reticulum Ca²⁺-ATPase complex with CrATP. Journal of Biological Chemistry, 267(5), 3539-3550.

Vilsen, B. & Andersen, J. P. (1988). Ca2+ occlusion in monomeric sarcoplasmic reticulum Ca-ATPase. Progress in Clinical and Biological Research, 273, 147-154.

Vilsen, B., Andersen, J. P. & MacLennan, D. H. (1991). Functional consequences of alterations to amino acids located in the hinge domain of the Ca²⁺-ATPase of sarcoplasmic reticulum. Journal of Biological Chemistry, 266(24), 16157-16164.

Vilsen, B., Andersen, J. P., Clarke, D. M. & MacLennan, D. H. (1989). Functional consequences of proline mutations in the cytoplasmic and transmembrane sectors of the Ca²⁺-ATPase of sarcoplasmic reticulum. Journal of Biological Chemistry, 264(35), 21024-21030.

Vilsbøll, T., Zdravkovic, M., Le-Thi, T., Krarup, T., Schmitz, O., Courrèges, J.-P., Verhoeven, R., Bugànová, I. & Madsbad, S. (2007). Liraglutide, a long-acting human GLP-1 analog, given as monotherapy significantly improves glycemic control and lowers body weight without risk of hypoglycemia in patients with type 2 diabetes mellitus. Diabetes Care, 30(6), 1608-10.

Vilsbøll, T., Brock, B., Perrild, H., Levin, K., Lervang, H. H., Kølendorf, K. E., Krarup, T., Schmitz, O., Zdravkovic, M., Le-Thi, T. & Madsbad, S. (2008). Liraglutide, a once-daily human GLP-1 analogue, improves pancreatic B-cell function and arginine-stimulated insulin secretion during hyperglycaemia in patients with Type 2 diabetes mellitus. Diabetic Medicine, 25(2), 152-6. https://doi.org/10.1111/j.1464-5491.2007.02333.x

Vils, S. R., Troldborg, A., Hvas, A.-M. & Thiel, S. (2023). Platelets and the Lectin Pathway of Complement Activation in Patients with Systemic Lupus Erythematosus or Antiphospholipid Syndrome. TH open, 7(2), e155-e167. https://doi.org/10.1055/a-2087-0314

Villalba, N., E., S., Garcia-Sacristán, A., Simonsen, U. & Prieto, D. (2007). Contribution of both Ca2+ entry and Ca2+ sensitization to the alpha 1-adrenergic vasoconstriction of rat penile small arteries. Am. J. Physiol. Heart Circ Physiol, 292(2), H1157-69.

Villalba, N., E., S., Simonsen, U. & Prieto, D. (2008). Rho kinase is involved in Ca2+ entry of rat penile small arteries. American Journal of Physiology: Heart and Circulatory Physiology, 294, H1923-32.

Villalba, N., Kun, A., Stankevicius, E. & Simonsen, U. (2010). Role for Tyrosine Kinases in Contraction of Rat Penile Small Arteries. Journal of Sexual Medicine, 7(6), 2086-95. https://doi.org/10.1111/j.1743-6109.2010.01788.x

Villadsen, L. S., Skov, L., Dam, T. N., Dagnæs-Hansen, F., Rygaard, J., Schuurman, J., Parren, P. W., van de Winkel, J. G. & Baadsgaard, O. (2007). In situ depletion of CD4(+) T cells in human skin by Zanolimumab. Archives of Dermatological Research, 298, 449-455.

Vilhjálmsson, B. J., Yang, J., Finucane, H. K., Gusev, A., Lindström, S., Ripke, S., Genovese, G., Loh, P.-R., Bhatia, G., Do, R., Hayeck, T., Won, H.-H., Kathiresan, S., Pato, M., Pato, C., Tamimi, R., Stahl, E., Zaitlen, N., Pasaniuc, B. ... Børglum, A. (2015). Modeling Linkage Disequilibrium Increases Accuracy of Polygenic Risk Scores. American Journal of Human Genetics, 97(4), 576-92. https://doi.org/10.1016/j.ajhg.2015.09.001

Vildbrad, M. D., Andersen, A., Holmboe, S., Ringgaard, S., Nielsen, J. M. & Nielsen-Kudsk, J. E. (2014). Acute effects of levosimendan in experimental models of right ventricular hypertrophy and failure. Pulmonary Circulation, 4(3), 511-519. https://doi.org/10.1086/677366

Vildbrad, M. D., Andersen, A., Andersen, T. K., Axelgaard, S., Holmboe, S., Andersen, S., Ringgaard, S. & Nielsen-Kudsk, J. E. (2015). Limitations and pitfalls in measurements of right ventricular stroke volume in an animal model of right heart failure. Physiological Measurement, 36(5), 925-937. https://doi.org/10.1088/0967-3334/36/5/925

Vila, I. K., Chamma, H., Steer, A., Saccas, M., Taffoni, C., Turtoi, E., Reinert, L. S., Hussain, S., Marines, J., Jin, L., Bonnefont, X., Hubert, M., Schwartz, O., Paludan, S. R., Van Simaeys, G., Doumont, G., Sobhian, B., Vlachakis, D., Turtoi, A. & Laguette, N. (2022). STING orchestrates the crosstalk between polyunsaturated fatty acid metabolism and inflammatory responses. Cell Metabolism, 34(1), 125-139.e8. https://doi.org/10.1016/j.cmet.2021.12.007

Vila, I. K., Guha, S., Kalucka, J., Olagnier, D. & Laguette, N. (2022). Alternative pathways driven by STING: From innate immunity to lipid metabolism. Cytokine and Growth Factor Reviews, 68, 54-68. https://doi.org/10.1016/j.cytogfr.2022.08.006

Viidik, A. (2009). Darwins evolutionslära ingår kanske i Guds plan. Läkartidningen, 106(41), 2629-31.

Viidik, A. (2009). Kommersiella "ta själv"-gentest allt hetare--men värdet är osäkert. Läkartidningen, 106(11), 742-3.

Viidik, A. (2009). Gränserna flytande för forskningsfusk: svårt att upptäcka falska och friserade data. Läkartidningen, 106(36), 2222-3.

Viidik, A. (2003). Telomerer är inte nyckeln till evigt liv. Läkartidningen, 100( 30-31).

Viidik, A. (2008). Carrel lyckades aldrig förlänga livet på sina bindvävsceller. Läkartidningen, 105(14), 929-30.

Viidik, A. (2008). Kan man bli »berusad« efter sin död? Läkartidningen, 105(45), 3215.

Viidik, A. (2009). Antioxidanter och åldrande – sista ordet inte sagt. Läkartidningen, 106(15-16), 1084.

Viidik, A. (2009). Antioxidanter motverkar fria radikalers nyttiga effekter. Läkartidningen, 106(28-29), 1808.

Viidik, A. (2009). Kan man leva längre om man fastar? Läkartidningen, 106(39), 2442. http://ltarkiv.lakartidningen.se/2009/temp/pda37038.pdf

Viidik, A. (2010). Den exceptionellt långa livslängdens komplexa genetik. Läkartidningen, 107(37), 2148-49. http://www.lakartidningen.se/07engine.php?articleId=14947

Viidik, A. (2010). Rikhaltigt supplement från Japan om åldrandets biologi. Läkartidningen, 107(39), 243. http://www.lakartidningen.se/07engine.php?articleId=15055

Viidik, A. (2010). Ett maskformigt bihang till nytta för människan. Läkartidningen, 107(45), 2829-30. http://www.lakartidningen.se/07engine.php?articleId=15373

Viidik, A. (2011). Den arabiska vetenskapens guldålder - rättelse och förtydligande. Läkartidningen, 108(21), 1199. http://www.lakartidningen.se/07engine.php?articleId=16568

Viidik, A. (2011). Återställd telomerasaktivitet gav minskad vävnadsdegeneration. Läkartidningen, 108(8), 388. http://www.lakartidningen.se/07engine.php?articleId=16016

Viidik, A. (2011). Åldrande med »komprimerad sjuklighet« ännu en hägring. Sjukligheten under ålderdomen har snarare expanderat. Läkartidningen, 108(14), 778-79. http://www.lakartidningen.se/07engine.php?articleId=16268

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Revised 21.08.2025